Psoriasis
| Subclass of | skin disease, autoimmune skin disease, disease  |
|---|---|
| Health specialty | dermatology |
| Possible treatment | ultraviolet light therapy |
| Risk factor | smoking, stress, obesity, Xerosis cutis |
| WordLift URL | http://data.medicalrecords.com/medicalrecords/healthwise/psoriasis |
| ICPC 2 ID | S91 |
| NCI Thesaurus ID | C3346 |
Psoriasis be a long-lasting, noncontagious autoimmune disease wey be characterized by patches of abnormal skin.[1][2] Dese areas be red, pink, anaa purple, dry, itchy, den scaly.[3][4] Psoriasis dey vary in severity from small localized patches to complete body coverage.[3] Injury to de skin fi trigger psoriatic skin changes at dat spot, wich be known as de Koebner phenomenon.[5]
De five main types of psoriasis be plaque, guttate, inverse, pustular, den erythrodermic.[2] Plaque psoriasis, dem sanso know as psoriasis vulgaris, dey make up about 90% of cases.[1] E typically dey present as red patches plus white scales on top.[1] Areas of de body most commonly affected be de back of de forearms, shins, navel area, den scalp.[1] Guttate psoriasis get drop-shaped lesions.[2] Pustular psoriasis dey present as small, noninfectious, pus-filled blisters.[6] Inverse psoriasis dey form red patches in skin folds.[2] Erythrodermic psoriasis dey occur wen de rash cam be very widespread wey fi develop from any of de oda types.[1] Fingernails den toenails be affected insyd chaw people plus psoriasis at sam point insyd time.[1] Dis fi include pits insyd de nails anaa changes insyd nail color.[1]
Dem generally dey think psoriasis to be a genetic disease wey be triggered by environmental factors.[3] If one twin get psoriasis, de oda twin be three times more likely to be affected if de twins be identical dan if dem be nonidentical.[1] Dis dey suggest say genetic factors predispose to psoriasis.[1] Symptoms often worsen during winter den plus certain medications, such as beta blockers anaa NSAIDs.[1] Infections den psychological stress sanso fi play a role.[2][3] De underlying mechanism dey involve de immune system wey dey react to skin cells.[1] Diagnosis dey typically base on de signs den symptoms.[1]
Der be no known cure give psoriasis, buh various treatments fi help control de symptoms.[1] Dese treatments dey include steroid creams, vitamin D3 cream, ultraviolet light, immunosuppressive drugs, such as methotrexate, den biologic therapies wey dey target specific immunologic pathways.[2] About 75% of skin involvement dey improve plus creams per.[1] De disease dey affect 2–4% of de population.[7] Men den women be affected plus equal frequency.[2] De disease fi begin at any age, buh typically dey start insyd adulthood.[2] Psoriasis be associated plus an increased risk of psoriatic arthritis, lymphomas, cardiovascular disease, Crohn's disease, den depression.[1] Psoriatic arthritis dey affect up to 30% of individuals plus psoriasis.[6]
De word "psoriasis" be from Greek ψωρίασις wey dey mean 'itching condition' anaa 'being itchy',[8] from psora 'itch', den -iasis 'action, condition'.
References
[edit | edit source]- 1 2 3 4 5 6 7 8 9 10 11 12 13 14 15 Boehncke WH, Schön MP (September 2015). "Psoriasis". Lancet. 386 (9997): 983–94. doi:10.1016/S0140-6736(14)61909-7. PMID 26025581. S2CID 208793879.
- 1 2 3 4 5 6 7 8 "Questions and Answers About Psoriasis". National Institute of Arthritis and Musculoskeletal and Skin Diseases. 12 April 2017. Archived from the original on 22 April 2017. Retrieved 22 April 2017.
- 1 2 3 4 Menter, Alan; Gottlieb, Alice; Feldman, Steven R.; Van Voorhees, Abby S.; Leonardi, Craig L.; Gordon, Kenneth B.; Lebwohl, Mark; Koo, John Y.M.; Elmets, Craig A.; Korman, Neil J.; Beutner, Karl R.; Bhushan, Reva (2008). "Guidelines of care for the management of psoriasis and psoriatic arthritis". Journal of the American Academy of Dermatology (in English). 58 (5): 826–850. doi:10.1016/j.jaad.2008.02.039.
- ↑ LeMone P, Burke K, Dwyer T, Levett-Jones T, Moxham L, Reid-Searl K (2015). Medical-Surgical Nursing. Pearson Higher Education AU. p. 454. ISBN 978-1-4860-1440-8. Archived from the original on 14 January 2023. Retrieved 8 May 2020.
- ↑ Ely JW, Seabury Stone M (March 2010). "The generalized rash: part II. Diagnostic approach". American Family Physician. 81 (6): 735–9. PMID 20229972. Archived from the original on 2 February 2014.
- 1 2 Jain S (2012). Dermatology: illustrated study guide and comprehensive board review. Springer. pp. 83–87. ISBN 978-1-4419-0524-6. Archived from the original on 8 September 2017.
- ↑ Parisi R, Symmons DP, Griffiths CE, Ashcroft DM (February 2013). "Global epidemiology of psoriasis: a systematic review of incidence and prevalence". The Journal of Investigative Dermatology. 133 (2). Identification and Management of Psoriasis and Associated ComorbidiTy (IMPACT) project team: 377–85. doi:10.1038/jid.2012.339. PMID 23014338.
- ↑ Ritchlin C, Fitzgerald I (2007). Psoriatic and Reactive Arthritis: A Companion to Rheumatology (1st ed.). Maryland Heights, MI: Mosby. p. 4. ISBN 978-0-323-03622-1. Archived from the original on 8 January 2017.
Read further
[edit | edit source]- Baker BS (2008). From Arsenic to Biologicals: A 200 Year History of Psoriasis. Beckenham UK: Garner. ISBN 978-0-9551603-2-5.
- "Guidelines for the assessment and management of psoriasis". U.S. National Guideline Clearinghouse. Archived from the original on 27 September 2013. Retrieved 26 July 2013.
- World Health Organization (2016). Global report on psoriasis. World Health Organization (WHO). hdl:10665/204417. ISBN 978-92-4-156518-9. Archived from the original on 29 February 2016.
External links
[edit | edit source]
- "Psoriatic arthritis". Genetics Home Reference.
- "Psoriasis". MedlinePlus. U.S. National Library of Medicine.
